rabbit polyclonal anti p akt 473 (Proteintech)
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rabbit polyclonal anti p akt 473
Rabbit Polyclonal Anti P Akt 473, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 522 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+polyclonal+anti+p+akt/Phospho-AKT1+(Ser473)+Antibody/pmc11727224-91-35-42
Average 96 stars, based on 522 article reviews
Rabbit Polyclonal Anti P Akt 473, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 522 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+polyclonal+anti+p+akt/Phospho-AKT1+(Ser473)+Antibody/pmc11727224-91-35-42
Average 96 stars, based on 522 article reviews
rabbit polyclonal anti p akt 473 - by Bioz Stars,
2026-10
96/100 stars
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Membrane:Article Title: TREM2-mediated Macrophage Glycolysis Promotes Skin Wound Angiogenesis via the Akt/mTOR/HIF-1α Signaling Axis. Article Snippet: Department of Dermatology, Traditional Chinese and Western Medicine Hospital of Wuhan, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430022, China Hubei Provincial Key Laboratory of Skin Infection and Immunity, Wuhan No. 1 Hospital, Wuhan 430022, China Department of Dermatology, Institute of Dermatology, Chinese Academy of Medical Sciences and Peking Union Medical College, Nanjing 210000, China Incubation:Article Title: TREM2-mediated Macrophage Glycolysis Promotes Skin Wound Angiogenesis via the Akt/mTOR/HIF-1α Signaling Axis. Article Snippet: Department of Dermatology, Traditional Chinese and Western Medicine Hospital of Wuhan, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430022, China Hubei Provincial Key Laboratory of Skin Infection and Immunity, Wuhan No. 1 Hospital, Wuhan 430022, China Department of Dermatology, Institute of Dermatology, Chinese Academy of Medical Sciences and Peking Union Medical College, Nanjing 210000, China Article Title: TREM2 alleviates long-term cognitive dysfunction after subarachnoid hemorrhage in mice by attenuating hippocampal neuroinflammation via PI3K/Akt signaling pathway. Article Snippet: Subarachnoid hemorrhage (SAH) often leads to long-term cognitive deficits in patients, particularly due to injury to brain regions such as the hippocampus.. This study aims to investigate the role of the triggering receptor expressed on myeloid cells 2 (TREM2) in mitigating hippocampal injury and associated cognitive impairments following SAH.. To explore the protective effects of TREM2, we utilized the TREM2 agonist COG1410 to upregulate TREM2 expression and employed TREM2 knockout (KO) mice to verify the necessity of TREM2 for this protective role. Article Title: Prevotella copri transplantation promotes neurorehabilitation in a mouse model of traumatic brain injury Article Snippet: The sample proteins (20 μg/lane) were separated by sodium dodecyl sulfate–polyacrylamide gel electrophoresis (SDS‒PAGE) (Invitrogen) and transferred onto polyvinylidene uoride (PVDF) membranes (Millipore, Boston, MA, USA). .. Membranes were blocked with 5% nonfat milk for 1 hour at room temperature and then incubated overnight at 4°C with primary antibodies, including rabbit monoclonal anti-β-actin (1:5000, ab213262, Abcam), rabbit polyclonal antiZO-1 (1:1000, 21773-1-AP, Proteintech), rabbit polyclonal anti-Occludin (1:1000, 27260-1-AP, Proteintech), rabbit monoclonal anti-Bcl-2 (1:1000, ab182858, Abcam), rabbit monoclonal anti-Bax (1:1000, ab182733, Abcam), Article Title: Prevotella copri transplantation promotes neurorehabilitation in a mouse model of traumatic brain injury Article Snippet: The sample proteins (20 μg/lane) were separated by sodium dodecyl sulfate–polyacrylamide gel electrophoresis (SDS‒PAGE) (Invitrogen) and transferred onto polyvinylidene fluoride (PVDF) membranes (Millipore, Boston, MA, USA). .. Membranes were blocked with 5% nonfat milk for 1 h at room temperature and then incubated overnight at 4 °C with primary antibodies, including rabbit monoclonal anti-β-actin (1:5000, ab213262, Abcam), rabbit polyclonal anti-ZO-1 (1:1000, 21773-1-AP, Proteintech), rabbit polyclonal anti-Occludin (1:1000, 27260-1-AP, Proteintech), rabbit monoclonal anti-Bcl-2 (1:1000, ab182858, Abcam), rabbit monoclonal anti-Bax (1:1000, ab182733, Abcam), Article Title: Inhibition of TREM-1 ameliorates Lipopolysaccharide-induced depressive-like behaviors by alleviating neuroinflammation in the PFC via PI3K/Akt signaling pathway. Article Snippet: Neuroinflammation is closely related to depression and is a key pathophysiological process of depression.. Triggering receptor expressed on myeloid cells 1 (TREM-1) has been proven to exert proinflammatory effects in various diseases.. However, the role of TREM-1 in depression has not been elucidated. Article Title: TREM2 activation alleviates neural damage via Akt/CREB/BDNF signalling after traumatic brain injury in mice Article Snippet: The sample proteins (20 μg/lane) were separated by sodium dodecyl sulfate–polyacrylamide gel electrophoresis (SDS-PAGE) (Invitrogen) and transferred onto polyvinylidene fluoride (PVDF) membranes (Millipore, Boston, MA, USA). .. Membranes were blocked with 5% nonfat milk for 1 h at room temperature, then incubated overnight at 4 °C with primary antibodies, including rabbit monoclonal anti-β-actin (1:5000, Cat# ab213262, Abcam), rabbit monoclonal anti-TREM2 (1:1000, Cat# 91068S, Cell Signaling Technology), rabbit Polyclonal anti-ZO-1 (1:1000, Cat# 21773-1-AP, Proteintech), rabbit Polyclonal anti-Occludin (1:1000, Cat# 27260-1-AP, Proteintech), rabbit Polyclonal anti-Claudin-5 (1:500, Cat# 34-1600, ThermoFisher), rabbit Polyclonal anti-TNF-α (1:1000, Cat# 17590-1-AP, Proteintech), rabbit monoclonal anti-IL-1β (1:1000, Cat# ab254360, Abcam), rabbit monoclonal anti-Bcl-2 (1:1000, Cat# ab182858, Abcam), rabbit monoclonal anti-Bax (1:1000, Cat# ab182733, Abcam), rabbit Polyclonal anti-Caspase-3 (1:1000, Cat# 19677-1-AP, Proteintech), |